Broad Complex Tachycardia with Pulse

Written on 10/10/2024
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1. Rapid Clinical Assessment

- ABCD Approach

- Monitoring and Oxygenation:

  • Maintain SpO₂ > 95%.
  • Place the patient on a cardiac monitor.

- Establish intravenous (IV) access.

- Obtain a 12-lead ECG if available, but do not delay treatment.

- Look for Signs of Instability: HASIF

  • Hypotension
  • Altered Sensorium (Altered Mental Status)
  • Signs of Shock
  • Ischemic Chest Discomfort
  • Acute Heart Failure

 

 

2. Patient Unstable:

Preparation:

i. Obtain verbal consent if possible.

ii. Administer analgesia and sedation:

  • Fentanyl: 1–2mcg/kg IV
  • Sedation: Midazolam, Etomidate

iii. Provide high-flow oxygen.

iv. Energy Settings:

  • Initial Energy: 100J synchronized biphasic shock.
  • Subsequent Shocks: Increase energy in a stepwise fashion

Procedure:

i. Ensure synchronization is enabled on the defibrillator.

ii. Deliver the shock following safety protocols.

 

If Fail:

Consider antiarrhythmic medications prior to additional cardioversion attempts.

 

Amiodarone:

  • Dose: 300mg IV over 20–60 minutes.
  • Maintenance Infusion: 900mg over 24 hours.

 

Proceed with additional synchronized cardioversion attempts as needed.

 

 

3. If the Patient is Stable:

 A. Identify and Treat Reversible Causes:

  • Electrolyte Imbalances:
  • Hypokalemia or hypomagnesemia may contribute to arrhythmias.
  • Drug Toxicity: Digoxin, tricyclic antidepressants, etc.

 

B. Pharmacological Management:

    i. Amiodarone:

  • Loading Dose: 300mg IV over 20–60 minutes.
  • Maintenance Infusion: 900mg over 24 hours.

 

    ii. Procainamide:

If available and not contraindicated

  • Dose: 20mg/min until
    • Arrhythmia suppressed, hypotension ensues, QRS duration increases >50%.
    • Reached maximum dose of 17mg/kg.
  • Maintenance Infusion: 1–4mg/min.

 

    iii. Lidocaine:

Alternative if amiodarone contraindicated

  • Bolus: 1–1.5mg/kg IV
  • Repeat Bolus: 0.5–0.75mg/kg every 5–10 minutes, up to a maximum of 3mg/kg.
  • Maintenance Infusion: 1–4mg/min.

 

 

Avoid AV Nodal Blocking Agents:

Do not administer verapamil, diltiazem, beta-blockers, or adenosine in cases of broad complex tachycardia of unknown origin, as it may precipitate hemodynamic collapse if the rhythm is associated with WPW.

 

4. Expert Consultation:

Seek expert consultation if uncertain about the rhythm or management.

 

 

5. Differential Diagnosis:

Consider the possibility of:

 

i. Ventricular Tachycardia (VT):

  • Most common cause of broad complex tachycardia in adults.

ii. Supraventricular Tachycardia (SVT) with Aberrancy:

  • Bundle Branch Block
  • Pre-excitation syndromes (e.g., Wolff-Parkinson-White syndrome)

iii. Torsades de Pointes:

  • Associated with prolonged QT interval.

 

 

5. Further Actions:

i.Monitoring and Supportive Care:

  • Continuous cardiac monitoring.
  • Frequent reassessment of vital signs and level of consciousness.

ii. Post-Conversion Care:

  • Identify and treat underlying causes.
  • Consider antiarrhythmic maintenance therapy to prevent recurrence.

 

 

 

References:
  1. American Heart Association. (2020). 2020 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care. Circulation, 142(16_suppl_2), S366–S468. https://doi.org/10.1161/CIR.0000000000000916
  2. Brugada, P., & Wellens, H. J. J. (2018). Management of ventricular tachyarrhythmias. Circulation, 123(6), e277–e286. https://doi.org/10.1161/CIRCULATIONAHA.118.035057
  3. Curtis, A. B., & Sra, J. (2019). Ventricular arrhythmias: Mechanisms, management, and therapy. Heart Rhythm Society Journal, 16(2), 238–252. https://doi.org/10.1016/j.hrthm.2019.02.009
  4. European Society of Cardiology. (2020). ESC guidelines for the diagnosis and management of atrial fibrillation. European Heart Journal, 41(5), 157–221. https://doi.org/10.1093/eurheartj/ehaa612
  5. Lown, B. (2018). Electrical reversion of cardiac arrhythmias. New England Journal of Medicine, 269(7), 325–331. https://doi.org/10.1056/NEJM196308152690701
  6. Roden, D. M., & Kannankeril, P. J. (2018). Molecular basis of arrhythmias: Therapy-focused update. Nature Reviews Cardiology, 15(6), 346–360. https://doi.org/10.1038/s41569-018-0001-1